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  Herpes simplex Infection

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Articles published in PLoS Pathog

Retrieve available abstracts of 16 articles:
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    January 2017
  1. DEMBOWSKI JA, Dremel SE, DeLuca NA
    Replication-Coupled Recruitment of Viral and Cellular Factors to Herpes Simplex Virus Type 1 Replication Forks for the Maintenance and Expression of Viral Genomes.
    PLoS Pathog. 2017;13:e1006166.
    PubMed     Text format     Abstract available

    September 2016
  2. SAWTELL NM, Thompson RL
    De Novo Herpes Simplex Virus VP16 Expression Gates a Dynamic Programmatic Transition and Sets the Latent/Lytic Balance during Acute Infection in Trigeminal Ganglia.
    PLoS Pathog. 2016;12:e1005877.
    PubMed     Text format     Abstract available

  3. MAROUI MA, Calle A, Cohen C, Streichenberger N, et al
    Latency Entry of Herpes Simplex Virus 1 Is Determined by the Interaction of Its Genome with the Nuclear Environment.
    PLoS Pathog. 2016;12:e1005834.
    PubMed     Text format     Abstract available

    June 2016
  4. RUSSELL TA, Tscharke DC
    Lytic Promoters Express Protein during Herpes Simplex Virus Latency.
    PLoS Pathog. 2016;12:e1005729.
    PubMed     Text format     Abstract available

    April 2016
  5. CONN KL, Hendzel MJ, Schang LM
    Correction:The Differential Mobilization of Histones H3.1 and H3.3 by Herpes Simplex Virus 1 Relates Histone Dynamics to the Assembly of Viral Chromatin.
    PLoS Pathog. 2016;12:e1005575.
    PubMed     Text format     Abstract available

    March 2016
  6. WILCOX DR, Longnecker R
    The Herpes Simplex Virus Neurovirulence Factor gamma34.5: Revealing Virus-Host Interactions.
    PLoS Pathog. 2016;12:e1005449.
    PubMed     Text format    

    July 2015
  7. ROSATO PC, Leib DA
    Neuronal Interferon Signaling Is Required for Protection against Herpes Simplex Virus Replication and Pathogenesis.
    PLoS Pathog. 2015;11:e1005028.
    PubMed     Text format     Abstract available

    June 2015
  8. FUNK C, Ott M, Raschbichler V, Nagel CH, et al
    The Herpes Simplex Virus Protein pUL31 Escorts Nucleocapsids to Sites of Nuclear Egress, a Process Coordinated by Its N-Terminal Domain.
    PLoS Pathog. 2015;11:e1004957.
    PubMed     Text format     Abstract available

    May 2015
    Selective Recruitment of Nuclear Factors to Productively Replicating Herpes Simplex Virus Genomes.
    PLoS Pathog. 2015;11:e1004939.
    PubMed     Text format     Abstract available

    April 2015
  10. BAGDONAITE I, Norden R, Joshi HJ, Dabelsteen S, et al
    A Strategy for O-Glycoproteomics of Enveloped Viruses-the O-Glycoproteome of Herpes Simplex Virus Type 1.
    PLoS Pathog. 2015;11:e1004784.
    PubMed     Text format     Abstract available

  11. KIM M, Truong NR, James V, Bosnjak L, et al
    Relay of Herpes Simplex Virus between Langerhans Cells and Dermal Dendritic Cells in Human Skin.
    PLoS Pathog. 2015;11:e1004812.
    PubMed     Text format     Abstract available

    March 2015
  12. RAMAKRISHNA C, Ferraioli A, Calle A, Nguyen TK, et al
    Establishment of HSV1 latency in immunodeficient mice facilitates efficient in vivo reactivation.
    PLoS Pathog. 2015;11:e1004730.
    PubMed     Text format     Abstract available

    January 2015
  13. CABRERA JR, Viejo-Borbolla A, Martinez-Martin N, Blanco S, et al
    Secreted Herpes Simplex Virus-2 Glycoprotein G Modifies NGF-TrkA Signaling to Attract Free Nerve Endings to the Site of Infection.
    PLoS Pathog. 2015;11:e1004571.
    PubMed     Text format     Abstract available

  14. WILCOX DR, Wadhwani NR, Longnecker R, Muller WJ, et al
    Differential reliance on autophagy for protection from HSV encephalitis between newborns and adults.
    PLoS Pathog. 2015;11:e1004580.
    PubMed     Text format     Abstract available

    December 2014
  15. GOODE D, Truong R, Villegas G, Calenda G, et al
    HSV-2-Driven Increase in the Expression of alpha4beta7 Correlates with Increased Susceptibility to Vaginal SHIVSF162P3 Infection.
    PLoS Pathog. 2014;10:e1004567.
    PubMed     Text format     Abstract available

    March 2014
  16. NDJAMEN B, Farley AH, Lee T, Fraser SE, et al
    The herpes virus Fc receptor gE-gI mediates antibody bipolar bridging to clear viral antigens from the cell surface.
    PLoS Pathog. 2014;10:e1003961.
    PubMed     Text format     Abstract available

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