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Articles published in PLoS Genet

Retrieve available abstracts of 8 articles:
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Single Articles

    May 2018
  1. FUNIKOV SY, Kulikova DA, Krasnov GS, Rezvykh AP, et al
    Spontaneous gain of susceptibility suggests a novel mechanism of resistance to hybrid dysgenesis in Drosophila virilis.
    PLoS Genet. 2018;14:e1007400.
    PubMed     Text format     Abstract available

    March 2018
  2. MANTEROLA M, Brown TM, Oh MY, Garyn C, et al
    BRDT is an essential epigenetic regulator for proper chromatin organization, silencing of sex chromosomes and crossover formation in male meiosis.
    PLoS Genet. 2018;14:e1007209.
    PubMed     Text format     Abstract available

    November 2017
  3. DUNLEAVY JEM, Okuda H, O'Connor AE, Merriner DJ, et al
    Katanin-like 2 (KATNAL2) functions in multiple aspects of haploid male germ cell development in the mouse.
    PLoS Genet. 2017;13:e1007078.
    PubMed     Text format     Abstract available

    August 2017
  4. JAIN D, Meydan C, Lange J, Claeys Bouuaert C, et al
    rahu is a mutant allele of Dnmt3c, encoding a DNA methyltransferase homolog required for meiosis and transposon repression in the mouse male germline.
    PLoS Genet. 2017;13:e1006964.
    PubMed     Text format     Abstract available

  5. SKOV L, Schierup MH
    Analysis of 62 hybrid assembled human Y chromosomes exposes rapid structural changes and high rates of gene conversion.
    PLoS Genet. 2017;13:e1006834.
    PubMed     Text format     Abstract available

    May 2017
  6. SHARMA V, Pandey AK, Kumar A, Misra S, et al
    Functional male accessory glands and fertility in Drosophila require novel ecdysone receptor.
    PLoS Genet. 2017;13:e1006788.
    PubMed     Text format     Abstract available

    April 2017
  7. WANG YY, Lin YH, Wu YN, Chen YL, et al
    Loss of SLC9A3 decreases CFTR protein and causes obstructed azoospermia in mice.
    PLoS Genet. 2017;13:e1006715.
    PubMed     Text format     Abstract available

    March 2017
  8. SHEN YR, Wang HY, Kuo YC, Shih SC, et al
    SEPT12 phosphorylation results in loss of the septin ring/sperm annulus, defective sperm motility and poor male fertility.
    PLoS Genet. 2017;13:e1006631.
    PubMed     Text format     Abstract available

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